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Ever been confused by all the overwhelming information and different strategies to cure premature ejaculation? Let me show you the top 5 misleading suggestions I've uncovered on the web. Many common 'solutions' to prevent premature ejaculation are completely counterproductive. Anyone who is familiar with my writing or my work as a sexual healer will understand what I mean. To successfully treat premature ejaculation or increase sexual stamina it is important to experience a sexual moment deeply and completely. Many of the premature ejaculation remedies available today come from the opposite perspective. Let's run through a few of these techniques that take you further away from sexual fulfillment and satisfaction. 1. Numbing Creams / Sprays / Extra condoms Numb says it all. What's the point of sex if you're not really feeling it. Your partner doesn't want a human vibrator. Trust me, there's a time for toys and there's a time for a real live man. I don't want mine with extra layers of latex and dead nerve endings thankyou very much. When I have a man inside me I want to know he is feeling it! Plus, I've heard that that those numbing potions can affect the woman during sex. Imagine that, two people going through the motions without feeling it! 2. Distracting Yourself A lot of folklore about male sexual performance revolves around the idea of delaying orgasm by distracting yourself. Thinking about your mother-in-law or sports, biting your cheek etc are all methods I have heard of or read about at some time or another. Turning off your arousal temporarily is not the same as mastering it. And again, if you only able to have sex by thinking about boring or distasteful things, how much fun is it going to be? This is a bad habit to get into. You don't want to train yourself to get bored and distracted when you are in the middle of hot lovin'. No woman is ever going to be interested in a man who can't focus on the matter at hand. Especially if she figures out you are thinking about your mother-in-law!! 3. Masturbation This is an interesting one. Remember that scene in 'Something About Mary' where he 'clears the pipes' before his big date to avoid seeming desperate. I have read that it is a good thing before sex to have a pre-emptive orgasm in order to delay the main event. I'd like to examine this a bit closer to see where this isn't helpful and maybe highlight when it is. Anxiety and stress about the impending sexual experience, thinking about the possibility of embarrassing yourself by coming too soon and focusing on the negatives of your sexual performance WHILE you masturbate is extremely unhelpful. Building a regime of this can be very damaging. It will have the effect of eroticising and reinforcing these aspects. You will associate orgasm even more with the stress and performance anxiety that is already a problem. However masturbating as part of a relaxed regime of self-pleasure and self-love can be very helpful. As long as your masturbation is not tied to your sexual performance it can be an enriching part of your sex life. In particular using self-pleasure to more fully explore your sexual arousal levels, orgasms, and control is the first step to deepening your awareness of sex. 4. Muscle Control Some techniques are like shaolin kung-fu disciplines that prescribe pelvic floor exercises. The theory is that with enough muscle strength and control you can prevent ejaculation escaping. By catching it with intense muscle contractions! Pretty spectacular stuff hey? In reality it's a bit like shutting the gate after the horse has bolted. Wouldn't you rather devote your time to exercises that help you understand your arousal levels so you can control the orgasm beforehand, and not just the fluid after the fact. And by the way, from what I gather it's extremely difficult to achieve anyway. 5. Alcohol / Drugs Relying on alcohol or drugs before you initiate a sexual encounter is a definite no no. Alcohol ultimately depresses your nervous system. And so depresses your ability to feel and maintain an erection. It is certainly the last thing you want to rely on for great sex. It may seem that you might loosen up your inhibitions temporarily. From the point of view of sexual stamina, forget it! And drugs - recreational or therapeutic. Some may give the illusion of increased sexual performance. But ultimately it is your level of awareness, your level of consciousness, your level of presence as a man that makes all the difference. I don't want you to think I'm a complete prude. This stuff isn't terrible in and of itself. I like the odd glass of champagne as much as the next girl. But my point is this. Using any or all of these techniques as the basis of your premature ejaculation cure will fail. It could actually make the situation worse. Relying on these things will lead you further from your true goal of sexual mastery and control of your orgasms. The only way to become a master of your sexuality is to go deeper into your sexual experiences, not further away. Learn about your body and orgasmic arousal by focusing on them. Don't shut your sexuality down in the quest for the 'appearance' of sexual mastery. A man with a numb penis, thinking about sports, thrusting for an hour, is about as far from a master as you can get. Love, Mukee Okan Copyright 2005 Mukee Okan penis enhancement surgery photo penis enlargement doctor enargement manhattan penis enlargement manhattan penis surgeon penis enlargment pill magna rx pnis enlargement result penis elargement technique vimax pillss inch

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Mums to be can often get worried about having sex while pregnant; well there is no medical evidence that having sexual intercourse whilst pregnant does any damage at all. There are a few cases where your GP may advise you about not having intercourse: - If you are Prone to miscarriages your GP may advise avoiding intercourse for the first three months or at the times when you would have been having your period when your hormones would be at their lowest. - You may be advised to avoid intercourse in the later stages of pregnancy if you have a history of premature labour - Your GP may advise you to avoid intercourse if you have a low lying placenta There is no need to worry about your baby being harmed by the penetration of the penis as it is well protected. This protection comes from the Muscular wall of the uterus, from the mucus plug that seals the neck of the uterus, and from the bag of waters. Sexual intercourse will not start labour if the body is not ready. However, if your baby is overdue, arousal of the nipple and intercourse could help in starting labour. The prostaglandins in semen soften the cervix, and hormones released by nipple stimulation encourage the uterus to contract. Sexual desires during pregnancy differ from person to person. Some women may just feel too ill or tired for sex, or a man may worry about harming the unborn child. Some women may just not feel sexually attractive whilst pregnant. In these cases it is best to talk as a couple to reassure each other of the others feelings. On the other hand many couples feel that pregnancy can do wonders for the sex life. Many men may be aroused by the fuller breasts and rounder hips of a pregnant partner. Some women feel great about not having to worry about contraceptives and periods. Well beyond all of that is the actual partaking in sexual intercourse where some people often worry. Of course as the pregnancy develops, the missionary position becomes impossible, which on a plus side forces into exploring other ways to make love. A few ideas are: - Woman on top – this not only gives the man a great view, but also lets the woman stay in control of position and penetration. - Rear entry positions – take time to find a position that is comfortable for you these rear entry positions also allow the man more penetration - Spoons – this is where both partners lye on their side and the man penetrates from behind, this gives shallower penetration which is more comfortable for some ladies during the later stages of pregnancy penis elargement excercises penis enargement drug best penis enlargement surgery penis elargement video natural penis enlargment pills homemade penile enlargement penis enhancement traction device homemade penis enlagement safe penile enlargment

Secure data backup has always been an issue for business. Let alone the fact that we live in a technologically-advanced society: most of us have not changed since the times when people used to keep their valuable items wrapped up in a bundle, hidden in their house. Take the data backup organization of companies and data centric organizations, for example. When the Bunsfield Oil Depot in Hemel Hempstead, Hertfordshire exploded in the morning of 11 December 2005, the truth about data backup and restoration became a serious issue. The huge explosions demolished all surrounding buildings, which could be heard at a radius of a hundred miles (160 km). The storage tanks were extinguished in two days, just to re-ignite on the 13th of December. The whole store was then left to burn to the ground. The unconfined vapor cloud explosion was another problem. The report claims that the vast explosion could be heard as far away as France and the Netherlands. There were over 700 companies within a 10- mile radius of the explosion. You can imagine exactly what happened to their data files, stored on tapes somewhere in a defeated safes. Fire-proof saves can really be a bargain, but only if the ruins of the falling building have not covered the said safe! After an explosion of such a rank, for example, this method of backup will prove its incompatibility to today’s perils that lay ahead of some companies. What if a building falls down, as happened to an IT managed services company waiting to transport its data offsite. The safe could not be uncovered, and the whole information was lost. A bankruptcy is what happens after such incidents, thinking about a logical sequence of events. The company was forced to issue a statement admitting its ability to service its customers had been "temporarily affected", with backup systems rendered inoperable. This was just an example, posed to illustrate the real dangers of irrational data backup, which many companies use to consider “safe”. Even more dangerous accidents can happen to a company, destroying all its mission critical files and data. Let’s involve some statistics. 75% of Companies Believe Their Data Is Safe if They Backup to Tape. They leave their valuable information unsafe and rely on procedures such as tape backup. Tapes can be stolen, destroyed, sold to rivals. Anything can happen to tapes. 97% of Companies Believe Their Data Is Safe if They Backup to Tape and Store the Media Offsite. Offsite media storage is no longer enough; the information is not safe, if the backup tapes are destroyed by accident. Backup to Tape is No Longer Enough. The worst thing is that the highly threatened companies still don’t realize it. Pay attention that Buncefield was only the 5th largest of over 50 refinery, storage and distribution sites in the UK, all of which are situated near major motorways and conurbations. The question that you should pose to your mind is whether you can afford to be struck by a disaster. The tape method of backup is no longer totally safe. The solution we offer to you is called Offsite Backup: an automatic backup of your data at two remote data centers in different countries, which have redundant connections between themselves. As your data is secured in two different countries, the whole information can be accessed any time at any day that you choose. The replication of your data in two different centers is a real solution to all your security bothers. PerfectBackup offers a simple, affordable and secure way to backup all your files: Exchange, SQL, MySQL, Oracle and Lotus data, ensuring the whole data set will always be available. For further details about our service, please visit www.perfectbackup.co.uk. Our site will be a serious guideline to all your questions. PerfectBackup is the solution offering nothing but benefits: it combines the strong points of tape and offsite backup, eliminating the traditional drawbacks. It will protect your data a secure offsite location, and make you more self-assertive about the further enlargement of your company. natural penis enhancement and lengthening penile enlargement surgery herbal penis elargement pnis girth enlargement best penile enlargement guide to penis enhancement penis enargement traction device penile enlargment system safe penile enlargment

If you’ve ever witnessed someone suffer a stroke, you understand the humbling nature of this disease. It can reduce the mightiest human being to an immobile, helpless creature. Impairment of crucial functions like speech, walking, and control of bowel and bladder can wrench control from the body in a moment. Even perpetually youthful TV personality Dick Clark was struck down by stroke at age 75, despite the outward appearance of perfect health. Clark’s stroke resulted in a six-week hospital stay and, judging from fragmented reports, significant disability. Stroke can be like a devastating fire that strikes without warning, leaving only smoldering rubble. Stroke can so ravage basic bodily functions that often all you can hope for is to regain a portion through rehabilitation. The disease process that underlies stroke requires decades—30 or 40 years—to develop. With that much lead time, why aren’t we better able to detect or stop this crippling disease? The truth is that we are able to predict many, if not most, strokes. Advances in imaging technology allow detection of atherosclerotic plaque that cause stroke years before it becomes a threat. Progress in deciphering the causes of stroke has also leapt forward. Unfortunately, your neighborhood physician still focuses on diagnosing the crisis rather than anticipating it. Physicians prefer to deal with catastrophes and are just not that interested in prevention. Most physicians ask: “Is it time to operate or not?” The medical community obsesses over procedures like carotid endarterectomy (surgical removal of plaque) or carotid stents. Even when a person is afforded the warnings of a “mini-stroke”, or transient ischemic attack (TIA), little more is done once it’s determined that surgery is not necessary—even though this person has high risk for future stroke (50% over 10 years). Let’s flip-flop this approach to stroke. Procedures represent a failure of prevention! Where do strokes come from? Stroke develops when some portion of the brain is deprived of blood. This usually results from a tiny bit of debris that dislodges from an atherosclerotic plaque along the walls of an artery (the same sort that accumulates in coronaries causing heart attack). The sources of debris have been a subject of controversy, but new imaging technologies have settled the question. Any blood vessel that leads from the heart to the brain can be a source. The two carotid arteries on both sides of your neck are a frequent source, as these arteries are prone to develop plaque. (Our discussion will be confined to what are called thromboembolic, or ischemic, strokes, i.e, strokes that occur from plaque that fragments, sending debris to the brain, and will not include the far less common hemorrhagic strokes due to rupture of small vessels in the brain, nor will we discuss atrial fibrillation and other heart causes of stroke. The thromboembolic strokes we discuss cause around 88% of all strokes.) Over the last 10 years, the aorta has been recognized as another important source of stroke. The aorta is the main artery of the body whose branches go to the head, arms, and legs. Atherosclerotic plaque is a live tissue that, through poor diet, inactivity, high cholesterol, overweight, etc., grows and becomes progressively more unstable. At some point, plaque fragments. Little bits break away, traveling to the brain. Fractured plaque also exposes its deeper structures to flowing blood, triggering blood clot formation, which in turn can also fragment and go to the brain. Atherosclerotic plaque is a prerequisite for the most common causes of stroke. If the majority of strokes originate from plaque, why not measure plaque to determine if you’re at risk for stroke? How can we easily, safely, and accurately measure plaque in the carotid arteries and aorta? And if plaque can be measured, can it be shrunk or inactivated to reduce or eliminate risk for stroke? How can plaque be measured? Just 20 years ago, the only practical method of identifying plaque in the carotids or aorta was through angiography, requiring catheters inserted into the body to inject x-ray dye. Angiography was impractical as a screening measure. CT scanning and magnetic resonance imaging (MRI) are emerging as exciting methods of imaging both carotids and aorta. Unfortunately, most centers and physicians are much more focused on the diagnostic uses of these technologies for people who have already suffered stroke or other catastrophe, and application of these devices for preventive uses is still evolving. One exception is when aortic calcification or aortic enlargement is incidentally noted on the increasingly popular CT heart scans; this is an important finding that can signal presence of aortic plaque. The one test that is widely available and can be performed in just about any center is carotid ultrasound. It’s simple, painless, and precise. Two basic observations can be made: 1. Plaque detection—Atherosclerotic plaque can be clearly visualized. If plaque blocks more than 70% of the diameter of the vessel, or if there are “soft” (unstable) elements in plaque, then stroke risk may be high enough to justify surgery or stents. However, if there are plaques that are less severe, substantial risk for stroke may still be present that can be reduced with preventive measures. 2. Carotid intimal-medial thickness—This is a measure of the thickness of the lining of the carotid artery in areas not involved by plaque, but often precedes the development of mature plaque. Carotid intimal-medial thickness also provides an index of body-wide potential for atherosclerotic plaque that can place you at risk for stroke. The aorta, for instance, cannot be well imaged by surface ultrasound but can still be a source for stroke. Increased carotid intimal-medial thickness and carotid plaque are closely associated with likelihood of aortic plaque. The Rotterdam Study of 4000 participants demonstrated that if carotid intimal-medial thickness is greater than normal (1.0 mm), then you can be at risk for stroke (and heart attack), even if no carotid plaques are detected. Carotid ultrasound is the one test you should consider that provides the most information with least effort. Ultrasound is harmless, painless, and can be obtained just about anywhere. Even if your doctor disagrees with your request for a carotid ultrasound, an increasing number of mobile services are popping up nationwide that make this test available for around $100. One important point: many scanners and interpreters will only report whether plaque is present or not. While this is important information, you should request that the carotid-intimal medial thickness be made as well. Not all centers can make this simple measure (because of software requirements), but it doesn’t hurt to try. Any amount of carotid plaque is reason to follow a preventive program, even if the plaque is insufficient to justify surgery. Can plaque be reduced? Can we shrink plaque in carotid arteries and aorta and thereby reduce, perhaps eliminate, these sources of stroke? That question is gaining momentum as effective therapies become available that pack real punch for reducing plaque. Study after study has now documented that plaque can be reduced and, with it, risk for stroke. Reduction in plaque of 10–20% is possible within a year or two. Let’s consider the most potent influences on carotid and aortic plaque growth that need to be considered in a plaque-reducing program. (I assume that you are a non-smoker—if you are a smoker, you first need to concentrate on quitting.) Hypertension Considerable experience documents the power of blood pressure-lowering for prevention of stroke. The most recently updated guidelines, the JNC–VII, recommends a blood pressure of 407 mg/dl heightens stroke risk six-fold. C-reactive protein (CRP) This measure of inflammation is proving to be a useful marker for identifying people at risk for stroke, with increased risk beginning at a level of 0.5 mg/l. High CRP also predicts more rapidly growing carotid plaque. Homocysteine Homocysteine is an important marker of increased likelihood of both carotid and aortic plaque, as well as stroke. In 1997, the European Concerted Action Project reported more than a doubling of stroke when homocysteine levels exceeded 12 mol/l. As homocysteine increases to 20 μmol/l, risk for stroke and heart attack increases an amazing 10-fold over that at a level of 9 μmol/l. Asymmetric dimethylarginine (ADMA) ADMA is recently discovered amino acid whose blood levels can skyrocket up to 10-fold in the presence of hypertension, metabolic syndrome, diabetes, high cholesterol and triglycerides, obesity, and high homocysteine levels. ADMA blocks the action of the amino acid, l-arginine. This mimicry reduces the availability of nitric oxide, a powerful dilator and protector of arteries. ADMA levels in the top 10% predict a six-fold heightened risk for future stroke, and ADMA levels in people with strokes are double that in other people. A carotid ultrasound study in 116 subjects showed that higher blood levels of ADMA are associated with more severe carotid plaque. Because of ADMA’s shared role across a variety of abnormal conditions, correction or blocking the action of ADMA has been suggested as a unique therapeutic tool to reduce stroke risk. Cholesterol Data suggest that lowering cholesterol with statin cholesterol-lowering drugs slows carotid plaque growth and reduce stroke risk approximately 22%. An interesting study from the Cardiovascular Institute at Mt. Sinai School of Medicine in New York using the precise measuring ability of MRI of the carotids and thoracic aorta showed an impressive 20% regression of plaque area with simvastatin (Zocor®) taken for two years. Although guidelines for cholesterol treatment recommend reduction of LDL cholesterol to 100 mg/dl in high-risk persons, a report from the Walter Reed Army Medical Center in Washington, DC, showed that carotid plaque was more effectively reduced when LDL cholesterol of 70 mg/dl or lower was achieved with statin cholesterol drugs. Lower LDL cholesterol may, therefore, be better. Treatment Strategies to Reduce Carotid and Aortic Plaque The essential question: How do we reduce carotid and aortic plaque? If we make this the focus of our efforts, many pieces begin to fall into place. If you’ve had any measure of carotid or aortic plaque such as a carotid ultrasound or aortic calcification on a CT heart scan, you know that you’re at increased risk for stroke. You also have a baseline for future comparison to gauge whether your program is working or not. Because most people have not one but several causes of carotid and aortic plaque, there is no one single treatment that effectively eliminates risk for stroke. Instead, most people require a comprehensive program of healthy diet, exercise, supplements, and medication when indicated. Here, we focus on the nutritional supplements that can be critical components of your plaque-reduction program. Fish oil Fish oil is a cornerstone of your stroke prevention program. Epidemiological observations suggest a strong relationship of fish intake and reduction of stroke risk. Carotid ultrasound studies demonstrate less carotid plaque with greater intakes of fish. A cleverly designed University of Southampton study made the fascinating observation that fish oil transforms the structure of carotid plaque. 150 people with severe carotid plaque scheduled for carotid endarterectomy (surgical removal of the plaque) were given fish oil, sunflower oil, or no treatment over several months while waiting for their procedure. (Delays in the British health system permitted this unique design.) Plaque was removed at surgery and examined. Participants taking fish oil had reduced inflammation in plaque and thicker tissue covering the fatty core, markers of more stable plaque. Those taking sunflower oil or no treatment had unstable plaques with greater inflammation and thinner, less sturdy covering tissue. This suggests that fish oil stabilizes carotid plaque, making it less likely to rupture and fragment. A standard capsule of fish oil (containing 300 mg of EPA + DHA) contains the same amount of omega-3s as a 3 oz serving of cod or halibut; three capsules (900 mg DHA + EPA) contain the equivalent of a serving of farm-raised salmon. The dose that seems to provide greatest protection from stroke, lowers triglycerides (that form abnormal lipoproteins; see above), and reduces fibrinogen, is four capsules per day (1200 mg EPA + DHA). Coenzyme Q10 (CoQ10) Although there are no data specifically addressing whether CoQ10 reduces plaque, it is a marvelously effective way to reduce blood pressure, one of the crucial factors causing carotid and aortic plaque growth. A pooled analysis of eight studies showed that, on average, CoQ10 in daily doses of 50–200 mg reduced systolic blood pressure by 16 mm Hg, diastolic pressure by 10 mm Hg. Data suggest that CoQ10 can reverse abnormal heart muscle thickening (hypertrophy), another manifestation of high blood pressure, strongly suggesting that CoQ10 has benefits beyond just reducing pressure. Supplements to correct the metabolic syndrome Weight loss is, without question, the most immediate and direct path to correction of this dangerous pre-diabetic condition. A drop of even 10–20 lbs yields improvements across the board: increased sensitivity to insulin, increased HDL, and reductions in triglycerides, CRP, fibrinogen, small LDL particles, and blood pressure. Diet and exercise are fundamental components of an effort to lose weight; low carbohydrate or reduced glycemic index diets (e.g., South Beach or Mediterranean) rich in fibers are clearly effective. Several supplements can amplify weight-reduction efforts and be useful adjuncts to your lifestyle program. Among them: White bean extract White bean extract blocks intestinal absorption of carbohydrates by 66%. 1500 mg twice a day with meals yields, on average, 3–7 lbs of weight loss in the first month of use. The only side-effect is excessive gas, due to unabsorbed starches. Glucomannan This unique fiber taken prior to meals absorbs many times its weight in water and thereby fills your stomach. You consequently take in less food. Most people lose around four lbs per month using 1500 mg prior to each meal. Interestingly, glucomannan also blunts the rise in blood sugar after meals, an effect that, by itself, may lead to weight loss. Be sure to take with plenty of water. DHEA This adrenal hormone is key to maintaining physical stamina, mood, muscle mass in men, and libido in women. A recent randomized, placebo-controlled study at Washington University in 56 subjects showed a 13% decline in abdominal fat (fat that drives resistance to insulin) measured by MRI with 50 mg of DHEA per day at bedtime, along with improved sugar control and lower insulin levels. Pectin, beta-glucan Pectin is the soluble fiber in citrus rinds, green vegetables, and apples, also available as a supplement. Beta-glucan is the soluble fiber of oats and is also available as a supplement. Both are wonderful fibers that provide feelings of fullness, lower cholesterol, slow release of sugars, and can yield modest weight reduction. A USC study in 573 subjects using carotid ultrasound showed that greater intake of healthy fibers like pectin and beta-glucan is associated with less carotid plaque growth. Folic acid, vitamins B6 and B12 Dr. Daniel Hackam at the Stroke Prevention and Atherosclerosis Research Centre in Ontario conducted a study using carotid ultrasound in 101 participants treated with folic acid 2.5 mg, vitamin B6 25 mg, and B12 250 mcg per day. Treatment resulted in plaque reduction, especially when homocysteine levels exceeded 14μmol/l at the start, compared to untreated participants who experienced substantial plaque growth. An attempt to clarify the role of homocysteine treatment was made through a National Institute of Health-sponsored study of stroke prevention. 3680 participants with a prior history of stroke were enrolled and given either a “low-dose” (20 mcg folic acid, 0.2 mg B6, 6 mcg B12) or a “high-dose” (2.5 mg folic acid, 25 mg B6, 400 mcg B12) regimen. Although starting homocysteine levels showed a graded association with stroke risk (higher homocysteine levels predicted greater stroke risk), the treatment groups experienced, on average, only a 2 μmol drop in homocysteine levels and no reduction in stroke risk over two years. The study investigators as well as critics have suggested that the study failed due to an insufficient treatment period and that the doses were too low. (The doses we use in our plaque reduction program are folic acid 2.5–5.0 mg, B6 50–100 mg, B12 1000–2500 mcg.) L-arginine L-arginine can be used to overpower the adverse effects of ADMA. L-arginine is emerging as an important carotid plaque-reversing tool. Early reports in animals showed that l-arginine completely halted growth of aortic plaque, and did so more effectively than lovastatin (a cholesterol-lowering drug). In humans, L-arginine reduces blood pressure, abnormal constriction of carotid and coronary arteries, blocks entry of inflammatory cells into plaque, increases sensitivity to insulin, and heightens exercise capacity. Following coronary angioplasty or stent placement, l-arginine results in up to 36% reduction in plaque growth. The average American takes in 5400 mg of l-arginine through food every day. Supplementing with doses of 3000–12,000 mg per day has proven useful to correct many of these phenomena. (We use a dose of 6000 mg of l-arginine powder, twice a day on an empty stomach, dissolved in water, for our plaque regression program.) Does this result in a reduction of stroke risk? The emerging data suggest that l-arginine is likely to exert a powerful plaque-reducing and stroke-preventing benefit, but we await more clinical trial data. Conclusion Reducing stroke risk by reversing carotid and aortic plaque is becoming an everyday reality, with better tools becoming available. To know whether you’re at risk, the best and most available imaging tool is carotid ultrasound, aiming to identify intimal-medial thickness >1.0 mm, or carotid plaque. Any degree of calcification of the aorta, such as on a CT heart scan, is another useful measure of risk. Treatment to reduce risk is multi-faceted but is based on examining all your sources of risk, including metabolic syndrome, small LDL, lipoprotein(a), and C-reactive protein. Fish oil is the one absolutely crucial ingredient in any stroke prevention program. Other supplements can be used in a targeted fashion, depending on the causes identified for your carotid or aortic plaque. Ideally, repeat scanning of your carotids should be done sometime after your program has begun to assess whether you’ve successfully achieved reversal of plaque growth. surgical penile enlargement pennis enlargement procedure manual penis enargement real penis enlagement penis elargement information vimax penis pills in uk top pennis enlargement pills medical pnis enlargement safe penile enlargment

I’m beginning to get the feeling that I’ve been spending too much time online. Just the other day I was leafing through the TV listings only to find myself analysing the keyword density of the film reviews. I have noticed other strange things going on as well. Next door to where I live is a church which has a row of small square windows on one side of the building. Every time I see those windows I think they are thumbnail images which I can click to open up larger images. What on earth is wrong with me? When I’m driving down the road and I see company adverts on the side of vans and trucks I think to myself: “Well that’s not very well optimised for search engine rankings!” Have I gone mad? Or could there be another explanation? Every profession has what is commonly referred to as an occupational hazard. Tennis players get tennis elbow, writers get writer’s cramp and politicians get liar’s teeth. For web designers and SEO professionals the problem seems to be one of not being able to tell the difference between the Internet and the Outernet (formerly known as the ‘real world’). A visit to my local optician confirmed that I had indeed gone Google-eyed. I was advised to seek help and enrol on a 12-step SEO rehabilitation program. Of course initially I denied that I had a problem and tried to claim that I just needed a few more incoming links and all would be fine. But when the hallway from your bedroom to your bathroom becomes a hypertext link, you know you’ve got problems. And when you return home one day to find the door to your apartment is locked and a message pops up in your head asking you to enter your username and password, you know it’s time to stand up and say: “My name is Rob, I’m an SEO addict and I’ve been online for too long.” So what are the symptoms of going Google-eyed? There are a number of tell-tale signs, one of which is an obsession with the length of your little green bar. I am of course referring to your Google Page Rank. Luckily treatment is available for this condition and it involves a large dose or perspective and reality. So let’s get this ailment cleared up straight away: Having a high Google Page Rank does not necessarily mean your site will show up in the top ten search results for your chosen keywords. Page Rank is like your ego: it’s great if you want to show off to your mates but it has little value beyond that. If people can find your site and those people become customers or clients, then it really doesn’t matter how big your Page Rank is and you can stop making those secret nocturnal visits to websites offering you backstreet Page Rank enlargement services! Now that we’ve cleared that up, let’s continue with our treatment. The second set of symptoms of Google-Eyedness has to do with a phenomenon that I have already discussed, namely, that of confusing the inner and outer worlds. If you spend so much time online that your food cupboards have become as bare as a newborn baby’s behind and when you eventually do venture out for groceries you find yourself wondering why your local supermarket doesn’t have a search engine so you can find what you want and leave… TURN OFF YOUR COMPUTER and repeat the following mantra: There is more to life than Google. There is more to life than Google. There is more to life than Google. You can have self-esteem without Google Page Rank and if your website is not in Google’s top ten, it’s not the end of the world! You see there seems to be this misconception that Google is all-powerful. We all know how much of the market share it controls and we all know how much difference it can make to the income of an online business. But Google is only as important as you think it is and only as powerful as we allow it to be. It’s easy to fall into the trap of believing that you need a higher Page Rank or a higher position in Google’s search results when what you may be in need of is a better sales conversion rate. I know people who obsess about Page Rank and getting their website into Google’s top ten and yet when I look at their webstats I find that they are getting loads of visitors to their site – but they’re not turning those visitors into paying customers. If your store looks dull and uninspiring and your products are poorly displayed you won’t make more money by moving the store to a better location or by herding more people in through the door: you need to address the issue at hand and not get distracted into believing that the problem has anything to do with Google. So why am I saying all this and why haven’t I written one of those ‘Top Ten SEO Tips’ articles instead? (I know you love reading them just as much as I do.) Because, as much as we may think it important to gain the approval of Google, if we place too much importance on this and become Google-eyed, we run the risk of losing sight of all the other opportunities available to us on the Internet and of becoming blind to the all the other online marketing strategies at our disposal. Explaining what those opportunities and strategies are will have to wait for another day. For now I have to continue my rehabilitation by taking a holiday.